Phenotype Search Results

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These related phenotype terms may also be of interest: osmotic stress resistance .

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4 Single Mutant Phenotype(s) for hypoosmotic stress resistance
Gene Name Organism Experiment type Mutant Information Phenotype Details References All phenotypes
csmA/AN6318 A. nidulans classical genetics Description: null hypoosmotic stress resistance: decreased Condition: YG medium
Takeshita N, et al.  (2002) csmA, a gene encoding a class V chitin synthase with a myosin motor-like domain of Aspergillus nidulans, is translated as a single polypeptide and regulated in response to osmotic conditions. Biochem Biophys Res Commun 298(1):103-9
csmA phenotypes
hex1/AO090020000450 A. oryzae classical genetics Description: null hypoosmotic stress resistance: decreased Details: decreased resistance to hypotonic shock after flooding the plates with water
Lopatin PV and Tol'tsman TI  (1965) [Determination of the aseptic area of the ward]. Aptechn Delo 14(3):14-9
hex1 phenotypes
pex11-1/AO090003000168 A. oryzae classical genetics Description: null
Allele: pex11-1::adeA 		hypoosmotic stress resistance: decreased Details: hyphal tip bursting due to hypotonic shock induced by flooding the plate with water; Woronin body function was examined by the ability to retain cytoplasmic constituents in the second compartment upon differential interference contrast microscopy
Escano CS, et al.  (2009) Disruption of the Aopex11-1 gene involved in peroxisome proliferation leads to impaired Woronin body formation in Aspergillus oryzae. Eukaryot Cell 8(3):296-305
pex11-1 phenotypes
so/AO090003000023 A. oryzae classical genetics Description: null
Allele: so::adaA 		hypoosmotic stress resistance: decreased Condition: addition of water to the strains grown on agar medium to induce hypotonic shock and hyphal tip bursting
Details: excessive cytoplasmic leakage upon hyphal injury 		Maruyama J, et al.  (2010) AoSO protein accumulates at the septal pore in response to various stresses in the filamentous fungus Aspergillus oryzae. Biochem Biophys Res Commun 391(1):868-73
so phenotypes


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